Con pena y no sin dolor, Daniel de 20 años de edad, falleció el pasado 20 de Octubre; tuvo un infarto masivo........
Esta bebida está de venta en todos los supermercados de nuestro país.Nuestros hijos, seres queridos y amigos la pueden consumir para probarla...puede ser mortal.
Red Bull fue creado para estimular el cerebro en personas sometidas a un gran esfuerzo físico y nunca para ser consumido como una bebida inocente o refrescante.
RED BULL es la bebida que se comercializa a nivel mundial con su eslogan:
'Aumenta la resistencia física, agiliza la capacidad de concentración y la velocidad de reacción, brinda más energía y mejora el estado de ánimo.
Todo eso se puede encontrar en una latita de RED BULL, la bebida energizante del milenio. (según sus codiciosos propietarios).
RED BULL ha logrado llegar a casi 100 países de todo el mundo. La marca del Toro Rojo, tiene como principales consumidores a jóvenes y deportistas, dos segmentos atractivos que han sido cautivados por el estímulo que produce la bebida.
HISTORIA:
Esta bebida fue creada por Dietrich Mateschitz, un empresario de origen austriaco quien la descubrió por casualidad en un viaje de negocios a HONG KONG, cuando trabajaba para una empresa fabricante de cepillos de dientes.
El liquido basado en una fórmula que contiene cafeína y taurina, causaba furor en ese país; entonces pensó en el rotundo éxito que esta bebida tendría en Europa, donde todavía no existía este producto, además de ver una oportunidad de convertirse en empresario.
¡PERO LA VERDAD DE ESTA BEBIDA ES OTRA! En Francia y Dinamarca la acaban de prohibir por ser un cóctel de la muerte, debido a sus componentes de vitaminas mezcladas con GLUCURONOLACTONE, químico altamente peligroso, desarrollado por el Departamento de Defensa de los Estados Unidos, durantelos años 60 para estimular la moral de las tropas acantonadas en VIETNAM, el cual actuaba como una droga alucinógena que calmaba el estrés de la guerra. Pero sus efectos en el organismo fueron tan devastadores, y fue descontinuado ante el alto índice de casos de migrañas, tumores cerebrales y enfermedades del hígado que mostraron los soldados que lo consumieron.
Y a pesar de ello, en la lata de RED BULL tiene entre otros componentes:
GLUCURONALACTONE, catalogado médicamente como un estimulante.
Pero lo que NO DICE la lata de RED BULL, son las consecuencias de su consumo, que obligan a colocar una serie de
ADVERTENCIAS:
1. Es peligroso tomarlo si después no hace ejercicio físico, ya que su función energizante acelera el ritmo cardiaco y te puede ocasionar un INFARTO FULMINANTE.
2. Corres el peligro de sufrir una HEMORRAGIA CEREBRAL, debido a que el RED BULL contiene componentes que diluyen la sangre para que al corazón le cueste menos bombearla, y así poder hacer un esfuerzo físico con menos agotamiento.
3. Está prohibido mezclar el RED BULL con alcohol, porque la mezcla convierte la bebida en una 'bomba mortal' que ataca directamente el hígado, provocando que la zona afectada no se regenere nunca más.
4. Uno de los componentes principales del RED BULL es la vitamina B12, utilizada en medicina para recuperar a pacientes que se encuentran en coma etílico (coma producido por consumo de alcohol); es por ello que se produce la hipertensión y el estado de excitabilidad en el que te encuentras después de tomarlo, como si estuvieras en estado de embriaguez, es decir borracho, sin haber tomado ninguna bebida alcohólica.
5. El Consumo regular de RED BULL desencadena en la aparición de una serie de enfermedades nerviosas y neuronales irreversibles (no hay recuperación).
CONCLUSIÓN:
Es una bebida que debería prohibirse en el mundo entero.
Venezuela, República Dominicana, Puerto Rico y otros países del Caribe, ya están alertando a otras naciones, ya que la mezcla de esta bebida con alcohol crea una bomba de tiempo para el cuerpo humano, principalmente entre los adolescentes y adultos ignorantes por su poca experiencia.
Esta bebida se vende en supermercados y tiendas del país y nuestros hijos, la pueden consumir para probarla... puede ser mortal!!
Sunday, April 27, 2008
Friday, April 11, 2008
20 consejos para convertirse en escritor
Tomado de Libros en red
Esta es una recopilación de las 20 normas básicas y decisivas que todo aspirante a escritor deberá tener en cuenta si quiere llegar a ser un novelista de éxito y codearse con los best-sellers. Algunas nos las han hecho llegar y otras son fruto de nuestra propia experiencia editorial. Cuantas más pautas se dominen, más posibilidades tendrá un autor de destacarse en su nueva profesión.
Aquí van:
1. Lo primero: conoser vien la hortografia.
2. Cuide la concordancia, el cual son necesaria para que usted no caigan en aquello errores.
3. Ponga comas puntos signos de interrogación o dos puntos rayas siempre que corresponda si no poco se entienden las relaciones entre las palabras la jerarquía entre las ideas.
Y cuando, use los signos de: puntuación, póngalos; correctamente!.
4. Lo mejor es esquivar la reiteración de sonidos en la oración. La proposición es buscar una opción que no rime con lo dicho con antelación.
5. Evite las repeticiones, evitando así repetir y repetir lo que ya ha repetido reiteradamente.
6. Trate de ser claro; no use hieráticos, herméticos o errabundos gongorismos que puedan jibarizar las más enaltecidas ideas.
7. Imaginando, creando, planificando, un escritor no debe aparecer equivocándose, abusando de los gerundios. Tratando siempre, sobre todo, de no estar empezando una frase con uno.
8. Correcto para ser en la construcción, caer evite en trasposiciones.
9. Tome el toro por las astas, haga de tripas corazón y no caiga en refranes comunes. Calavera no chilla.
10. ¡Voto al chápiro!... creo a pies juntillas que deben evitarse las antiguallas que obscurecen el texto.
11. Si algún lugar es inadecuado en la frase para poner colgado un verbo, el final de un párrafo lo es.
12. ¡¡¡Por el amor de Dios!!!!, no abuse de las exclamaciones. NI de las Mayúsculas. Recuerde, además, que la cantidad de puntos suspensivos es siempre fija....... (¡solo tres!)
13. Pone cuidado en las conjugaciones cuando escribáis.
14. No utilice nunca doble negación.
15. Evite usar el adjetivo "mismo" como si fuera un pronombre; el mismo está para otra cosa.
16. Aunque se usen poco, es importante emplear los apóstrofo's correctamente.
17. No olvide poner las tildes que correspondan. Mas aun cuando es importante conocer cual es la significacion de una palabra, en caso de que haya una opcion con tilde y sin ella.
18. Procure "no poner" comillas "innecesariamente". No es un recurso para "resaltar" sino para "mencionar" una "voz ajena" al texto.
19. Procurar nunca los infinitivos separar demasiado.
20. Y con respecto a frases fragmentadas
Bueno... ya habrán visto que no son las 20 normas definitivas para pasar a ser escritor, aunque eso no quita que lo que sugieren pueda tenerse en cuenta con algún provecho. Queda la pregunta: ¿habrá consejos que de verdad sirvan para aprender a escribir mejor?
Monday, April 7, 2008
Alzheimer's Disease
Alzheimer's disease (AD), one form of dementia, is a progressive, degenerative brain disease. It affects memory, thinking, and behavior.
Alternative Names
Senile dementia/Alzheimer's type (SDAT)
Memory impairment is a necessary feature for the diagnosis of Alzheimer's or any type of dementia. Change in one of the following areas must also be present: language, decision-making ability, judgment, attention, and other areas of mental function and personality.
The rate of progression is different for each person. If AD develops rapidly, it is likely to continue to progress rapidly. If it has been slow to progress, it will likely continue on a slow course.
More than 4 million Americans currently have AD. The older you get, the greater your risk of developing AD, although it is not a part of normal aging. Family history is another common risk factor.
In addition to age and family history, risk factors for AD may include:
Longstanding high blood pressure
History of head trauma
High levels of homocysteine (a body chemical that contributes to chronic illnesses such as heart disease, depression, and possibly AD)
Female gender -- because women usually live longer than men, they are more likely to develop AD
There are two types of AD -- early onset and late onset. In early onset AD, symptoms first appear before age 60. Early onset AD is much less common, accounting for only 5-10% of cases. However, it tends to progress rapidly.
The cause of AD is not entirely known but is thought to include both genetic and environmental factors. A diagnosis of AD is made based on characteristic symptoms and by excluding other causes of dementia.
Prior theories regarding the accumulation of aluminum, lead, mercury, and other substances in the brain leading to AD have been disproved. The only way to know for certain that someone had AD is by microscopic examination of a sample of brain tissue after death.
The brain tissue shows "neurofibrillary tangles" (twisted fragments of protein within nerve cells that clog up the cell), "neuritic plaques" (abnormal clusters of dead and dying nerve cells, other brain cells, and protein), and "senile plaques" (areas where products of dying nerve cells have accumulated around protein). Although these changes occur to some extent in all brains with age, there are many more of them in the brains of people with AD.
The destruction of nerve cells (neurons) leads to a decrease in neurotransmitters (substances secreted by a neuron to send a message to another neuron). The correct balance of neurotransmitters is critical to the brain.
By causing both structural and chemical problems in the brain, AD appears to disconnect areas of the brain that normally work together.
About 10 percent of all people over 70 have significant memory problems and about half of those are due to AD. The number of people with AD doubles each decade past age 70. Having a close blood relative who developed AD increases your risk.
Early onset disease can run in families and involves autosomal dominant, inherited mutations that may be the cause of the disease. So far, three early onset genes have been identified.
Late onset AD, the most common form of the disease, develops in people 60 and older and is thought to be less likely to occur in families. Late onset AD may run in some families, but the role of genes is less direct and definitive. These genes may not cause the problem itself, but simply increase the likelihood of formation of plaques and tangles or other AD-related pathologies in the brain.
In the early stages, the symptoms of AD may be subtle and resemble signs that people mistakenly attribute to "natural aging." Symptoms often include:
Repeating statements
Misplacing items
Having trouble finding names for familiar objects
Getting lost on familiar routes
Personality changes
Losing interest in things previously enjoyed
Difficulty performing tasks that take some thought, but used to come easily, like balancing a checkbook, playing complex games (such as bridge), and learning new information or routines
In a more advanced stage, symptoms are more obvious:
Forgetting details about current events
Forgetting events in your own life history, losing awareness of who you are
Problems choosing proper clothing
Hallucinations, arguments, striking out, and violent behavior
Delusions, depression, agitation
Difficulty performing basic tasks like preparing meals and driving
At end stages of AD, a person can no longer survive without assistance. Most people in this stage no longer:
Understand language
Recognize family members
Perform basic activities of daily living such as eating, dressing, and bathing
The first step in diagnosing Alzheimer's disease is to establish that dementia is present. Then, the type of dementia should be clarified. A health care provider will take a history, do a physical exam (including a neurological exam), and perform a mental status examination.
Tests may be ordered to help determine if there is a treatable condition that could be causing dementia or contributing to the confusion of AD. These conditions include thyroid disease, vitamin deficiency, brain tumor, drug and medication intoxication, chronic infection, anemia, and severe depression.
AD usually has a characteristic pattern of symptoms and can be diagnosed by history and physical exam by an experienced clinician. Tests that are often done to evaluate or exclude other causes of dementia include computed tomography (CT), magnetic resonance imaging (MRI), and blood tests.
In the early stages of dementia, brain image scans may be normal. In later stages, an MRI may show a decrease in the size of the cortex of the brain or of the area of the brain responsible for memory (the hippocampus). While the scans do not confirm the diagnosis of AD, they do exclude other causes of dementia (such as stroke and tumor).
Treatment
Unfortunately, there is no cure for AD. The goals in treating AD are to:
Slow the progression of the disease.
Manage behavior problems, confusion, and agitation.
Modify the home environment.
Support family members and other caregivers.
The most promising treatments include lifestyle changes, medications, and antioxidant supplements like vitamin E and ginkgo biloba.
LIFESTYLE CHANGES
The following steps can help people with AD:
Walk regularly with a caregiver or other reliable companion. This can improve communication skills and prevent wandering.
Use bright light therapy to reduce insomnia and wandering.
Listen to calming music. This may reduce wandering and restlessness, boost brain chemicals, ease anxiety, enhance sleep, and improve behavior.
Get a pet dog.
Practice relaxation techniques.
Receive regular massages. This is relaxing and provides social interactions.
DRUG TREATMENT
Several drugs are available to try to slow the progression of AD and possibly improve the person's mental capabilities. Memantine (Namenda) is currently the only drug approved for the treatment of moderate-to-severe Alzheimer’s disease.
Other medicines include donepezil (Aricept), rivastigmine (Exelon), galantamine (Razadyne, formerly called Reminyl), and tacrine (Cognex). These drugs affect the level of a neurotransmitter in the brain called acetylcholine. They may cause nausea and vomiting. Tacrine also causes an elevation in liver enzymes and must be taken four times a day. It is now rarely used.
Aricept is taken once a day and may stabilize or even improve the person's mental capabilities. It is generally well tolerated. Exelon seems to work in a similar way. It is taken twice a day.
Other medicines may be needed to control aggressive, agitated, or dangerous behaviors. These are usually given in very low doses.
It may be necessary to stop any medications that make confusion worse. Such medicines may include pain killers, cimetidine, central nervous system depressants, antihistamines, sleeping pills, and others. Never change or stop taking any medicines without first talking to your doctor.
SUPPLEMENTS
Folate (vitamin B9) is critical to the health of the nervous system. Together with some other B vitamins, folate is also responsible for clearing homocysteine (a body chemical that contributes to chronic illnesses) from the blood. High levels of homocysteine and low levels of both folate and vitamin B12 have been found in people with AD. Although the benefits of taking these B vitamins for AD is not entirely clear, it may be worth considering them, particularly if your homocysteine levels are high.
Antioxidant supplements, like ginkgo biloba and vitamin E, scavenge free radicals. These products of metabolism are highly reactive and can damage cells throughout the body.
Vitamin E dissolves in fat, readily enters the brain, and may slow down cell damage. In at least one well-designed study of people with AD who were followed for 2 years, those who took vitamin E supplements had improved symptoms compared to those who took a placebo pill. Patients who take blood-thinning medications like warfarin (Coumadin) may should talk to their doctor before taking vitamin E.
Ginkgo biloba is an herb widely used in Europe for treating dementia. It improves blood flow in the brain and contains flavonoids (plant substances) that act as antioxidants. Although many of the studies to date have been somewhat flawed, the idea that ginkgo may improve thinking, learning, and memory in those with AD has been promising. DO NOT use ginkgo if you take blood-thinning medications like warfarin (Coumadin) or a class of antidepressants called monoamine oxidase inhibitors (MAOIs).
If you are considering any drugs or supplements, you MUST talk to your doctor first. Remember that herbs and supplements available over the counter are NOT regulated by the FDA.
SUPPORT AT HOME
Someone with AD will need support in the home as the disease worsens. Family members or other caregivers can help by trying to understand how the person with AD perceives his or her world. Simplify the patient's surroundings. Give frequent reminders, notes, lists of routine tasks, or directions for daily activities. Give the person with AD a chance to talk about their challenges and participate in their own care.
OTHER PRACTICAL STEPS
The person with AD should have their eyes and ears checked. If problems are found, hearing aids, glasses, or cataract surgery may be needed.
Those with AD may have particular dietary requirements such as:
Extra calories due to increased physical activity from restlessness and wandering.
Supervised meals and help with feeding. People with AD often forget to eat and drink, and can become dehydrated as a result.
The Safe Return Program, implemented by the Alzheimer's Association, requires that a person with AD wear in identification bracelet. If he or she wanders, the caregiver can contact the police and the national Safe Return office, where information about the person is stored and shared nationwide.
Eventually, 24-hour monitoring and assistance may be necessary to provide a safe environment, control aggressive or agitated behavior, and meet physiologic needs. This may include in-home care, nursing homes, or adult day care.
Support Groups
For additional information and resources for people with Alzheimer's disease and their caregivers, see Alzheimer's disease support groups.
Expectations (prognosis)
The probable outcome is poor. The disorder is usually progresses steadily. Total disability is common. Death normally occurs within 15 years, usually from an infection or a failure of other body systems.
Complications
Loss of ability to function or care for self
Bedsores, muscle contractures (loss of ability to move joints because of loss of muscle function), infection (particularly urinary tract infections and pneumonia), and other complications related to immobility during end-stages of AD
Falls and broken bones
Loss of ability to interact
Malnutrition and dehydration
Failure of body systems
Reduced life span
Harmful or violent behavior toward self or others
Abuse by an over-stressed caregiver
Side effects of medications
Calling Your Health Care Provider
Call your health care provider if someone close to you experiences symptoms of senile dementia/Alzheimer's type.
Call your health care provider if a person with this disorder experiences a sudden change in mental status. (A rapid change may indicate other illness.)
Discuss the situation with your health care provider if you are caring for a person with this disorder and the condition deteriorates to the point where you can no longer care for the person in your home.
Although there is no proven way to prevent AD, there are some practices that may be worth incorporating into your daily routine, particularly if you have a family history of dementia. Talk to your doctor about any of these approaches, especially those that involve taking a medication or supplement.
Consume a low-fat diet.
Eat cold-water fish (like tuna, salmon, and mackerel) rich in omega-3 fatty acids, at least 2 to 3 times per week.
Reduce your intake of linoleic acid found in margarine, butter, and dairy products.
Increase antioxidants like carotenoids, vitamin E, and vitamin C by eating plenty of darkly colored fruits and vegetables.
Maintain a normal blood pressure.
Stay mentally and socially active throughout your life.
Consider taking non-steroidal anti-inflammatory drugs (NSAIDs) like ibuprofen (Advil, Motrin), sulindac (Clinoril), or indomethacin (Indocin). Statin drugs, a class of medications normally used for high cholesterol, may help lower your risk of AD. Talk to your doctor about the pros and cons of using these medications for prevention.
In addition, early testing of a vaccine against AD is underway.
Rakel P. Conn ’s Current Therapy 2005. 57th ed. Philadelphia, Pa: Saunders; 2005.
Moore DP, Jefferson JW. Handbook of Medical Psychiatry. 2nd ed. St. Louis, Mo: Mosby; 2004.
Goetz CG, Pappert EJ. Textbook of Clinical Neurology. 2nd ed. Philadelphia, Pa: Saunders; 2003.
Alternative Names
Senile dementia/Alzheimer's type (SDAT)
Memory impairment is a necessary feature for the diagnosis of Alzheimer's or any type of dementia. Change in one of the following areas must also be present: language, decision-making ability, judgment, attention, and other areas of mental function and personality.
The rate of progression is different for each person. If AD develops rapidly, it is likely to continue to progress rapidly. If it has been slow to progress, it will likely continue on a slow course.
More than 4 million Americans currently have AD. The older you get, the greater your risk of developing AD, although it is not a part of normal aging. Family history is another common risk factor.
In addition to age and family history, risk factors for AD may include:
Longstanding high blood pressure
History of head trauma
High levels of homocysteine (a body chemical that contributes to chronic illnesses such as heart disease, depression, and possibly AD)
Female gender -- because women usually live longer than men, they are more likely to develop AD
There are two types of AD -- early onset and late onset. In early onset AD, symptoms first appear before age 60. Early onset AD is much less common, accounting for only 5-10% of cases. However, it tends to progress rapidly.
The cause of AD is not entirely known but is thought to include both genetic and environmental factors. A diagnosis of AD is made based on characteristic symptoms and by excluding other causes of dementia.
Prior theories regarding the accumulation of aluminum, lead, mercury, and other substances in the brain leading to AD have been disproved. The only way to know for certain that someone had AD is by microscopic examination of a sample of brain tissue after death.
The brain tissue shows "neurofibrillary tangles" (twisted fragments of protein within nerve cells that clog up the cell), "neuritic plaques" (abnormal clusters of dead and dying nerve cells, other brain cells, and protein), and "senile plaques" (areas where products of dying nerve cells have accumulated around protein). Although these changes occur to some extent in all brains with age, there are many more of them in the brains of people with AD.
The destruction of nerve cells (neurons) leads to a decrease in neurotransmitters (substances secreted by a neuron to send a message to another neuron). The correct balance of neurotransmitters is critical to the brain.
By causing both structural and chemical problems in the brain, AD appears to disconnect areas of the brain that normally work together.
About 10 percent of all people over 70 have significant memory problems and about half of those are due to AD. The number of people with AD doubles each decade past age 70. Having a close blood relative who developed AD increases your risk.
Early onset disease can run in families and involves autosomal dominant, inherited mutations that may be the cause of the disease. So far, three early onset genes have been identified.
Late onset AD, the most common form of the disease, develops in people 60 and older and is thought to be less likely to occur in families. Late onset AD may run in some families, but the role of genes is less direct and definitive. These genes may not cause the problem itself, but simply increase the likelihood of formation of plaques and tangles or other AD-related pathologies in the brain.
In the early stages, the symptoms of AD may be subtle and resemble signs that people mistakenly attribute to "natural aging." Symptoms often include:
Repeating statements
Misplacing items
Having trouble finding names for familiar objects
Getting lost on familiar routes
Personality changes
Losing interest in things previously enjoyed
Difficulty performing tasks that take some thought, but used to come easily, like balancing a checkbook, playing complex games (such as bridge), and learning new information or routines
In a more advanced stage, symptoms are more obvious:
Forgetting details about current events
Forgetting events in your own life history, losing awareness of who you are
Problems choosing proper clothing
Hallucinations, arguments, striking out, and violent behavior
Delusions, depression, agitation
Difficulty performing basic tasks like preparing meals and driving
At end stages of AD, a person can no longer survive without assistance. Most people in this stage no longer:
Understand language
Recognize family members
Perform basic activities of daily living such as eating, dressing, and bathing
The first step in diagnosing Alzheimer's disease is to establish that dementia is present. Then, the type of dementia should be clarified. A health care provider will take a history, do a physical exam (including a neurological exam), and perform a mental status examination.
Tests may be ordered to help determine if there is a treatable condition that could be causing dementia or contributing to the confusion of AD. These conditions include thyroid disease, vitamin deficiency, brain tumor, drug and medication intoxication, chronic infection, anemia, and severe depression.
AD usually has a characteristic pattern of symptoms and can be diagnosed by history and physical exam by an experienced clinician. Tests that are often done to evaluate or exclude other causes of dementia include computed tomography (CT), magnetic resonance imaging (MRI), and blood tests.
In the early stages of dementia, brain image scans may be normal. In later stages, an MRI may show a decrease in the size of the cortex of the brain or of the area of the brain responsible for memory (the hippocampus). While the scans do not confirm the diagnosis of AD, they do exclude other causes of dementia (such as stroke and tumor).
Treatment
Unfortunately, there is no cure for AD. The goals in treating AD are to:
Slow the progression of the disease.
Manage behavior problems, confusion, and agitation.
Modify the home environment.
Support family members and other caregivers.
The most promising treatments include lifestyle changes, medications, and antioxidant supplements like vitamin E and ginkgo biloba.
LIFESTYLE CHANGES
The following steps can help people with AD:
Walk regularly with a caregiver or other reliable companion. This can improve communication skills and prevent wandering.
Use bright light therapy to reduce insomnia and wandering.
Listen to calming music. This may reduce wandering and restlessness, boost brain chemicals, ease anxiety, enhance sleep, and improve behavior.
Get a pet dog.
Practice relaxation techniques.
Receive regular massages. This is relaxing and provides social interactions.
DRUG TREATMENT
Several drugs are available to try to slow the progression of AD and possibly improve the person's mental capabilities. Memantine (Namenda) is currently the only drug approved for the treatment of moderate-to-severe Alzheimer’s disease.
Other medicines include donepezil (Aricept), rivastigmine (Exelon), galantamine (Razadyne, formerly called Reminyl), and tacrine (Cognex). These drugs affect the level of a neurotransmitter in the brain called acetylcholine. They may cause nausea and vomiting. Tacrine also causes an elevation in liver enzymes and must be taken four times a day. It is now rarely used.
Aricept is taken once a day and may stabilize or even improve the person's mental capabilities. It is generally well tolerated. Exelon seems to work in a similar way. It is taken twice a day.
Other medicines may be needed to control aggressive, agitated, or dangerous behaviors. These are usually given in very low doses.
It may be necessary to stop any medications that make confusion worse. Such medicines may include pain killers, cimetidine, central nervous system depressants, antihistamines, sleeping pills, and others. Never change or stop taking any medicines without first talking to your doctor.
SUPPLEMENTS
Folate (vitamin B9) is critical to the health of the nervous system. Together with some other B vitamins, folate is also responsible for clearing homocysteine (a body chemical that contributes to chronic illnesses) from the blood. High levels of homocysteine and low levels of both folate and vitamin B12 have been found in people with AD. Although the benefits of taking these B vitamins for AD is not entirely clear, it may be worth considering them, particularly if your homocysteine levels are high.
Antioxidant supplements, like ginkgo biloba and vitamin E, scavenge free radicals. These products of metabolism are highly reactive and can damage cells throughout the body.
Vitamin E dissolves in fat, readily enters the brain, and may slow down cell damage. In at least one well-designed study of people with AD who were followed for 2 years, those who took vitamin E supplements had improved symptoms compared to those who took a placebo pill. Patients who take blood-thinning medications like warfarin (Coumadin) may should talk to their doctor before taking vitamin E.
Ginkgo biloba is an herb widely used in Europe for treating dementia. It improves blood flow in the brain and contains flavonoids (plant substances) that act as antioxidants. Although many of the studies to date have been somewhat flawed, the idea that ginkgo may improve thinking, learning, and memory in those with AD has been promising. DO NOT use ginkgo if you take blood-thinning medications like warfarin (Coumadin) or a class of antidepressants called monoamine oxidase inhibitors (MAOIs).
If you are considering any drugs or supplements, you MUST talk to your doctor first. Remember that herbs and supplements available over the counter are NOT regulated by the FDA.
SUPPORT AT HOME
Someone with AD will need support in the home as the disease worsens. Family members or other caregivers can help by trying to understand how the person with AD perceives his or her world. Simplify the patient's surroundings. Give frequent reminders, notes, lists of routine tasks, or directions for daily activities. Give the person with AD a chance to talk about their challenges and participate in their own care.
OTHER PRACTICAL STEPS
The person with AD should have their eyes and ears checked. If problems are found, hearing aids, glasses, or cataract surgery may be needed.
Those with AD may have particular dietary requirements such as:
Extra calories due to increased physical activity from restlessness and wandering.
Supervised meals and help with feeding. People with AD often forget to eat and drink, and can become dehydrated as a result.
The Safe Return Program, implemented by the Alzheimer's Association, requires that a person with AD wear in identification bracelet. If he or she wanders, the caregiver can contact the police and the national Safe Return office, where information about the person is stored and shared nationwide.
Eventually, 24-hour monitoring and assistance may be necessary to provide a safe environment, control aggressive or agitated behavior, and meet physiologic needs. This may include in-home care, nursing homes, or adult day care.
Support Groups
For additional information and resources for people with Alzheimer's disease and their caregivers, see Alzheimer's disease support groups.
Expectations (prognosis)
The probable outcome is poor. The disorder is usually progresses steadily. Total disability is common. Death normally occurs within 15 years, usually from an infection or a failure of other body systems.
Complications
Loss of ability to function or care for self
Bedsores, muscle contractures (loss of ability to move joints because of loss of muscle function), infection (particularly urinary tract infections and pneumonia), and other complications related to immobility during end-stages of AD
Falls and broken bones
Loss of ability to interact
Malnutrition and dehydration
Failure of body systems
Reduced life span
Harmful or violent behavior toward self or others
Abuse by an over-stressed caregiver
Side effects of medications
Calling Your Health Care Provider
Call your health care provider if someone close to you experiences symptoms of senile dementia/Alzheimer's type.
Call your health care provider if a person with this disorder experiences a sudden change in mental status. (A rapid change may indicate other illness.)
Discuss the situation with your health care provider if you are caring for a person with this disorder and the condition deteriorates to the point where you can no longer care for the person in your home.
Although there is no proven way to prevent AD, there are some practices that may be worth incorporating into your daily routine, particularly if you have a family history of dementia. Talk to your doctor about any of these approaches, especially those that involve taking a medication or supplement.
Consume a low-fat diet.
Eat cold-water fish (like tuna, salmon, and mackerel) rich in omega-3 fatty acids, at least 2 to 3 times per week.
Reduce your intake of linoleic acid found in margarine, butter, and dairy products.
Increase antioxidants like carotenoids, vitamin E, and vitamin C by eating plenty of darkly colored fruits and vegetables.
Maintain a normal blood pressure.
Stay mentally and socially active throughout your life.
Consider taking non-steroidal anti-inflammatory drugs (NSAIDs) like ibuprofen (Advil, Motrin), sulindac (Clinoril), or indomethacin (Indocin). Statin drugs, a class of medications normally used for high cholesterol, may help lower your risk of AD. Talk to your doctor about the pros and cons of using these medications for prevention.
In addition, early testing of a vaccine against AD is underway.
Rakel P. Conn ’s Current Therapy 2005. 57th ed. Philadelphia, Pa: Saunders; 2005.
Moore DP, Jefferson JW. Handbook of Medical Psychiatry. 2nd ed. St. Louis, Mo: Mosby; 2004.
Goetz CG, Pappert EJ. Textbook of Clinical Neurology. 2nd ed. Philadelphia, Pa: Saunders; 2003.
Thursday, April 3, 2008
The first Lady of France
Bill Bonner
The Daily Reckoning
http://www.youtube.com/watch?v=flmoa2dVOSU
One of the world’s most fascinating people is surely the first lady ofFrance, Carla Bruni. She dined with Queen Elizabeth II last week. Amid allthe glamorous guests, glitzy table setting and diamonds, it was she whosparkled most. What follows has no apparent connection to our beat – money. Nor does ithave any particular connection with anything. Still, women represent halfthe human population. As poets, we are fascinated by them. As philosopherswe are intrigued. And as economists, we cannot ignore them. Mr. Sarkozy’swife is a woman, but not just any woman; she seems to us to be a kind ofüberfemale. Almost unnaturally gifted, talented, beautiful and corrupt. The trouble with economics – one of them – is that economists are almostall male. And thus they are completely incapable of understanding themotives or methods of half the population they are meant to be studying.That is why we turn our attention to Carla. She is such an extreme exampleof womanhood, perhaps we can learn something. For readers who have not followed the French press, Ms. Bruni is anextraordinary subject. The French say she is a “croqueuse d’hommes” –which is to say, a man-eater. It was she, according to the press, who hadsuch an affair with Mick Jagger that it destroyed his long marriage tothat tough, smart and talented Texan, Jerry Hall. And it was she, too, whois said to have had affairs with Eric Clapton and Donald Trump. Then, shelived with a French philosopher, Jean-Paul Enthoven, until she took afancy to his son, Raphael, with whom she had a son, Aurelien. Nor was sheparticularly demeure about the whole thing; she wrote a song called“Raphael” describing how she had fallen madly in love and had great sexwith him. She is so “properly, old-fashionedly beautiful,” writes India Knight inthe TIMES of London, “with non-inbred aristocratic features and goodbones; so beautiful that she makes everyone else look like pudding.” Not only that, she speaks three or four languages...comes from afabulously rich family...and can sing. At the Queen’s state dinner,practically every one of the hundreds of guests had seen her naked photoin the tabloids the day before. Yet, she was reportedly the most composed,most confident person there. According to reports, she is irresistible at every level – intellectually,emotionally, sexually and artistically. What’s more she lives around thecorner from your editor’s Paris apartment. “I saw her go by last night,” said a neighbor on Saturday. “She had awhole squad of police with her.” “Why doesn’t she move into the Elysee Palace with her husband?” we wantedto know. “Apparently, she likes to keep her independence.” Madame Sarkozy is like no first lady France has ever seen. Nor is she likeany first lady any country has ever seen. She says she finds monogamy“boring.” She further observed that “love lasts a long time, but burningdesire – two to three weeks.” Here is the world’s most desirable woman –married to a cad. But she is the perfect political prop...a woman whoplays her role superbly – according to the local gossip, both in publicand in private – and who otherwise goes on her way and amuses herself. What do other women think of her? Around the corner, from what we hear,they barely let their husbands out of sight. Your editor is an old-fashioned fuddy duddy; he admits it. He turns histired eyes to politics, economics and finance – and sees only frauds andmountebanks. But when he comes home at night, he is charmed. In thenewspapers, he regards the chatter on the editorial pages as all lies andclaptrap; but at home he believes every fairy tale. When his brokerproposes a hedge fund with a leveraged portfolio of private equity deals,he practically laughs in his face. But when his priest promises himeverlasting life, what reason has he to doubt? And when his daughter says,“Oh Daddy...I just dropped by the office to say hello,” he’s delighted,scarcely noticing that she leaves with $50 to buy a new pair of bluejeans. There is a place for cynicism, he says to himself, and a place to believe.Poor Carla doesn’t seem to know the difference. Still, she lives so close by. Better to keep an open mind. Besides, saysyour editor remembers that he has a couple weeks of vacation coming up... Until tomorrow,
Thursday, March 6, 2008
Easily Overlooked Lesions Tied to Colon Cancer
By DENISE GRADY from the NYTimes
Published: March 5, 2008
An easily overlooked type of abnormality in the colon is the most likely type to turn cancerous, and is more common in this country than previously thought, researchers are reporting.
»
Prevalence of Nonpolypoid (Flat and Depressed) Colorectal Neoplasms in Asymptomatic and Symptomatic Adults (JAMA)
The findings come from a study of colonoscopy, in which a camera-tipped tube is used to examine the lining of the intestine. Generally, doctors search for polyps, abnormal growths that stick out from the lining and can turn into cancer. But another type of growth is much more dangerous, and harder to see because it is flat or depressed and similar in color to healthy tissue.
Japanese researchers became concerned about these flat lesions in the 1980s and ’90s, but studies here had mixed results and American doctors tended to think that flat growths were less common and less dangerous in the United States.
The new study, to be published Wednesday in the Journal of the American Medical Association, suggests otherwise.
Some doctors in this country were already alert to flat lesions, but the findings will pose a challenge to others, because it takes a trained and vigilant eye to see the growths and special techniques to remove them. The results also mean it is especially important that patients take the harsh laxatives that many dread in advance of the test. The flat lesions, hard to find even under the best conditions, will be impossible to see if any waste is left in the bowel.
Colon cancer is the second-leading cause of cancer death in the United States, after lung cancer, with about 154,000 new cases detected and 52,000 deaths a year. It is one of the few cancers that is totally preventable if precancerous growths are found and removed; it can also be cured with surgery alone if found early enough.
People who have just had a colonoscopy should not rush to schedule another one just to look for the flat growths, doctors said.
“I don’t think people have to panic that they’ve somehow been neglected and had poor care,” said Dr. David A. Rothenberger, deputy chairman of surgery at the University of Minnesota.
But he and other experts emphasized that people should see a doctor any time they have persisting symptoms that could indicate colon cancer, like rectal bleeding or a change in bowel habits — no matter how recently they had a colonoscopy. The test is highly reliable, but not perfect, doctors say.
Some doctors who perform colonoscopy just are not good at seeing flat lesions, but may improve with training and practice, said Dr. Douglas K. Rex, a gastroenterologist and professor of medicine at Indiana University.
“I think there are people who expect everything in there to be shaped like a golf ball,” he said. “It’s not.”
Dr. David Lieberman, chief of gastroenterology at Oregon Health and Science University, who wrote an editorial accompanying the study, said: “I think there will be some surprise. There has been in general some skepticism in the United States about how common flat and depressed lesions are and how important they are. So I think this study, coming from the United States and from a good group of investigators, will be a wake-up call to a lot of physicians and will prompt people to be looking for these lesions.”
The study, of 1,819 military veterans, mostly men, found that 9.35 percent had flat lesions, and those lesions were five times as likely as polyps to contain cancerous or precancerous tissue. Depressed or indented lesions were the least common but the most risky. Together, the flat or depressed lesions accounted for only 15 percent of the potentially cancerous growths found in the study, but were involved in half of the cancers. Once the doctors spotted the flat lesions, they sprayed a bluish dye on them to see their outlines better and remove them completely.
The first author of the study, Dr. Roy M. Soetikno of the Veterans Affairs Palo Alto Health Care System said, “The message for doctors is, Here is a large amount of data showing that these precursors of cancer, always believed to be a Japanese disease, are actually a disease here, and are important, because they are much more likely to be cancerous, and doctors need to spend the time to provide quality colonoscopy.”
The message to patients, Dr. Soetikno said, is that when preparing for colonoscopy, they must follow instructions to the letter and take the hated laxatives to make sure their bowels are empty so that doctors can see the lining.
If any waste remains, flat lesions will be buried by it. Studies have shown that in about a quarter of all colonoscopies, the bowel preparation is inadequate.
Dr. Rex said that male veterans tended to have more precancerous colon growths than other groups, so the rate of flat lesions in women or the general population might not be quite as high as those in the study.
Dr. Soetikno and his colleagues started an exchange program with doctors in Japan to learn their techniques for recognizing and removing the flat lesions.
American doctors should learn from overseas colleagues more often, Dr. Rothenberger said, adding, “We tend to get very smug about our abilities.”
The quality of colonoscopy has become a delicate issue, because an article in The New England Journal of Medicine in December 2006 found that some doctors were 10 times better than others at finding precancerous polyps. A major factor in their success was taking enough time to examine the colon thoroughly, as opposed to rushing through the procedure. Doctors who miss polyps would almost certainly miss flat lesions as well because they are harder to see. The new study underscores the need for careful examinations, because the flat lesions are more dangerous.
The study also raises doubts about whether “virtual colonoscopy,” performed by a CT scanner, will ever be able to take the place of the colonoscope inserted into the rectum, as many patients had hoped. The problem is that CT scans use X-rays to reveal shapes, and find polyps because they stick out. Flat lesions are unlikely to show up in such scans.
Studies show that from 0.3 percent to 0.9 percent of patients develop colon cancers within just a few years of having a colonoscopy and polyp removal — exactly what the procedure is supposed to prevent. Some doctors think that flat lesions, missed entirely during the colonoscopy or not fully removed, may account for some of these apparent failures.
Dr. Robert Smith, the director of screening for the American Cancer Society, said flat lesions were “a vexing issue” that had provoked a lot of arguments among doctors.
“This paper shows they’re more prevalent than we believed, and also quite serious with regard to the presence of features associated with an elevated risk of cancer,” Dr. Smith said.
The difficulty facing patients is how to be sure their doctors are doing a good job. Professional groups have issued guidelines about the best way to perform a colonoscopy, but they are recommendations, not rules. The groups also urge doctors to track their own success rates at finding precancerous growths to see how they measure up to standards, but even if they do keep track, the doctors do not have to share the data with anyone. And many people are loath to ask about it. The doctor wielding the scope is the last person most patients would want to offend.
“The patient really has no way to act as an informed consumer,” Dr. Smith said. “You can’t call up a facility and say, ‘By the way, is my doctor any good?’ or, ‘Tell me who the best one is.’ ”
He added: “For some physicians there is an expectation of trust, and it is offputting to have a patient request documentation of competence. However, some physicians know patients are hearing about these issues and are not offended by questions about performance and errors.”
Published: March 5, 2008
An easily overlooked type of abnormality in the colon is the most likely type to turn cancerous, and is more common in this country than previously thought, researchers are reporting.
»
Prevalence of Nonpolypoid (Flat and Depressed) Colorectal Neoplasms in Asymptomatic and Symptomatic Adults (JAMA)
The findings come from a study of colonoscopy, in which a camera-tipped tube is used to examine the lining of the intestine. Generally, doctors search for polyps, abnormal growths that stick out from the lining and can turn into cancer. But another type of growth is much more dangerous, and harder to see because it is flat or depressed and similar in color to healthy tissue.
Japanese researchers became concerned about these flat lesions in the 1980s and ’90s, but studies here had mixed results and American doctors tended to think that flat growths were less common and less dangerous in the United States.
The new study, to be published Wednesday in the Journal of the American Medical Association, suggests otherwise.
Some doctors in this country were already alert to flat lesions, but the findings will pose a challenge to others, because it takes a trained and vigilant eye to see the growths and special techniques to remove them. The results also mean it is especially important that patients take the harsh laxatives that many dread in advance of the test. The flat lesions, hard to find even under the best conditions, will be impossible to see if any waste is left in the bowel.
Colon cancer is the second-leading cause of cancer death in the United States, after lung cancer, with about 154,000 new cases detected and 52,000 deaths a year. It is one of the few cancers that is totally preventable if precancerous growths are found and removed; it can also be cured with surgery alone if found early enough.
People who have just had a colonoscopy should not rush to schedule another one just to look for the flat growths, doctors said.
“I don’t think people have to panic that they’ve somehow been neglected and had poor care,” said Dr. David A. Rothenberger, deputy chairman of surgery at the University of Minnesota.
But he and other experts emphasized that people should see a doctor any time they have persisting symptoms that could indicate colon cancer, like rectal bleeding or a change in bowel habits — no matter how recently they had a colonoscopy. The test is highly reliable, but not perfect, doctors say.
Some doctors who perform colonoscopy just are not good at seeing flat lesions, but may improve with training and practice, said Dr. Douglas K. Rex, a gastroenterologist and professor of medicine at Indiana University.
“I think there are people who expect everything in there to be shaped like a golf ball,” he said. “It’s not.”
Dr. David Lieberman, chief of gastroenterology at Oregon Health and Science University, who wrote an editorial accompanying the study, said: “I think there will be some surprise. There has been in general some skepticism in the United States about how common flat and depressed lesions are and how important they are. So I think this study, coming from the United States and from a good group of investigators, will be a wake-up call to a lot of physicians and will prompt people to be looking for these lesions.”
The study, of 1,819 military veterans, mostly men, found that 9.35 percent had flat lesions, and those lesions were five times as likely as polyps to contain cancerous or precancerous tissue. Depressed or indented lesions were the least common but the most risky. Together, the flat or depressed lesions accounted for only 15 percent of the potentially cancerous growths found in the study, but were involved in half of the cancers. Once the doctors spotted the flat lesions, they sprayed a bluish dye on them to see their outlines better and remove them completely.
The first author of the study, Dr. Roy M. Soetikno of the Veterans Affairs Palo Alto Health Care System said, “The message for doctors is, Here is a large amount of data showing that these precursors of cancer, always believed to be a Japanese disease, are actually a disease here, and are important, because they are much more likely to be cancerous, and doctors need to spend the time to provide quality colonoscopy.”
The message to patients, Dr. Soetikno said, is that when preparing for colonoscopy, they must follow instructions to the letter and take the hated laxatives to make sure their bowels are empty so that doctors can see the lining.
If any waste remains, flat lesions will be buried by it. Studies have shown that in about a quarter of all colonoscopies, the bowel preparation is inadequate.
Dr. Rex said that male veterans tended to have more precancerous colon growths than other groups, so the rate of flat lesions in women or the general population might not be quite as high as those in the study.
Dr. Soetikno and his colleagues started an exchange program with doctors in Japan to learn their techniques for recognizing and removing the flat lesions.
American doctors should learn from overseas colleagues more often, Dr. Rothenberger said, adding, “We tend to get very smug about our abilities.”
The quality of colonoscopy has become a delicate issue, because an article in The New England Journal of Medicine in December 2006 found that some doctors were 10 times better than others at finding precancerous polyps. A major factor in their success was taking enough time to examine the colon thoroughly, as opposed to rushing through the procedure. Doctors who miss polyps would almost certainly miss flat lesions as well because they are harder to see. The new study underscores the need for careful examinations, because the flat lesions are more dangerous.
The study also raises doubts about whether “virtual colonoscopy,” performed by a CT scanner, will ever be able to take the place of the colonoscope inserted into the rectum, as many patients had hoped. The problem is that CT scans use X-rays to reveal shapes, and find polyps because they stick out. Flat lesions are unlikely to show up in such scans.
Studies show that from 0.3 percent to 0.9 percent of patients develop colon cancers within just a few years of having a colonoscopy and polyp removal — exactly what the procedure is supposed to prevent. Some doctors think that flat lesions, missed entirely during the colonoscopy or not fully removed, may account for some of these apparent failures.
Dr. Robert Smith, the director of screening for the American Cancer Society, said flat lesions were “a vexing issue” that had provoked a lot of arguments among doctors.
“This paper shows they’re more prevalent than we believed, and also quite serious with regard to the presence of features associated with an elevated risk of cancer,” Dr. Smith said.
The difficulty facing patients is how to be sure their doctors are doing a good job. Professional groups have issued guidelines about the best way to perform a colonoscopy, but they are recommendations, not rules. The groups also urge doctors to track their own success rates at finding precancerous growths to see how they measure up to standards, but even if they do keep track, the doctors do not have to share the data with anyone. And many people are loath to ask about it. The doctor wielding the scope is the last person most patients would want to offend.
“The patient really has no way to act as an informed consumer,” Dr. Smith said. “You can’t call up a facility and say, ‘By the way, is my doctor any good?’ or, ‘Tell me who the best one is.’ ”
He added: “For some physicians there is an expectation of trust, and it is offputting to have a patient request documentation of competence. However, some physicians know patients are hearing about these issues and are not offended by questions about performance and errors.”
REMEDIO PARA LA TOS - INFORMACIÓN ÚTIL SOBRE EL VICK VAPO-RUB.
Durante una conferencia sobre Aceites Esenciales, nos comentaron cómo las plantas de los pies pueden absorber aceites. Su ejemplo consistió en colocar ajo en la planta de los pies y a los 20 minutos, ¡ya se podría percibir su sabor en la boca!Algunos hemos usado el Vick Vaporub durante muchos años como remedio para muchas cosas, desde labios cuarteados hasta dedos de los pies inflamados y muchas otras partes de la piel que se puede pelar en los pliegues. Pero nunca había escuchado sobre esto. Y no lo tomes en broma, porque funciona el 100% de las veces que se hace, aunque los científicos que lo descubrieron realmente no están seguros cómo sucede.
Para detener la tos nocturna en un niño (o de un adulto, quien fue que nos lo comentó personalmente), esparza Vick Vaporub generosamente sobre las plantas de los pies y luego cubra con medias. Aún la tos más persistente, fuerte y profunda se detendrá en el término de unos 5 minutos y se detendrá para dar muchas horas de alivio. Funciona el 100% de las veces que se hace y es más efectivo en los niños, que aún las más fuertes medicinas prescritas por los médicos. Además, es extremadamente calmante y reconfortante, mientras duermen profundamente.
Acababa de sintonizar en la radio en AM y escuché un señor hablando sobre porqué las medicinas para la tos en los niños podrían causar más daño que mejoría, y es debido a la composición química de sus fuertes componentes, así que decidí escucharlo. Fue muy sorprendente saber que fuera másefectivo que los medicamentos prescritos para que los niños tomaran en las horas de la noche, y que además tenía una efecto calmante en los niños enfermos mientras dormían profundamente. Mi esposa lo comprobó en sí misma en una ocasión en que tuvo una tos muy profunda, constante y persistente durante semanas y le funcionó en un 100%.
Ella me dijo que se sentía como si una cobija tibia la envolviera, paraba la tos en unos minutos y créanme, esta era una tos profunda (increíblemente desesperante) que no se calmaba ni por segundos, y esa noche durmió sin tospor muchas horas y así fue durante cada noche que lo utilizaba. Si usted tiene hijos o nietos, distribuya este mensaje. Y si usted se llegara a encontrar enfermo, compruébelo por usted mismo y se maravillarátotalmente de ver y sentir cómo funciona.
Para detener la tos nocturna en un niño (o de un adulto, quien fue que nos lo comentó personalmente), esparza Vick Vaporub generosamente sobre las plantas de los pies y luego cubra con medias. Aún la tos más persistente, fuerte y profunda se detendrá en el término de unos 5 minutos y se detendrá para dar muchas horas de alivio. Funciona el 100% de las veces que se hace y es más efectivo en los niños, que aún las más fuertes medicinas prescritas por los médicos. Además, es extremadamente calmante y reconfortante, mientras duermen profundamente.
Acababa de sintonizar en la radio en AM y escuché un señor hablando sobre porqué las medicinas para la tos en los niños podrían causar más daño que mejoría, y es debido a la composición química de sus fuertes componentes, así que decidí escucharlo. Fue muy sorprendente saber que fuera másefectivo que los medicamentos prescritos para que los niños tomaran en las horas de la noche, y que además tenía una efecto calmante en los niños enfermos mientras dormían profundamente. Mi esposa lo comprobó en sí misma en una ocasión en que tuvo una tos muy profunda, constante y persistente durante semanas y le funcionó en un 100%.
Ella me dijo que se sentía como si una cobija tibia la envolviera, paraba la tos en unos minutos y créanme, esta era una tos profunda (increíblemente desesperante) que no se calmaba ni por segundos, y esa noche durmió sin tospor muchas horas y así fue durante cada noche que lo utilizaba. Si usted tiene hijos o nietos, distribuya este mensaje. Y si usted se llegara a encontrar enfermo, compruébelo por usted mismo y se maravillarátotalmente de ver y sentir cómo funciona.
Tuesday, January 29, 2008
NUEVO CÁNCER DE MAMAS
(Autora desconocida)
Esta es una información a tener en cuenta por nosotras las mujeres. Creo que es importante que la divulguemos entre nuestras amigas.
En noviembre, la hermana de una amiga desarrolló una erupción en su mama, similar a la que sufren las madres jóvenes que están amamantando.
Debido a que su mamografía salió limpia, el doctor la trató con antibióticos contra infección.
Después de dos tratamientos, empezó a empeorar. Su médico le mandó hacer otra mamografía y esta vez se apreció una masa cuya biopsia reveló un tumor maligno de rápido crecimiento.
Primero le ordenaron quimioterapia para reducir el crecimiento. Luego siguió una mastectomía y después un tratamiento completo de quimioterapia, seguido de radiación.....
Nueve meses después de intenso tratamiento, fue dada de alta.
Después de un año de vivir cada día al máximo posible, el cáncer le tomó el hígado.
Siguió cuatro tratamientos y decidió, finalmente, que quería calidad de vida y no sufrir los efectos secundarios de la quimioterapia.
Tuvimos cinco extraordinarios meses y ella planeó todos y cada uno de los detalles de sus días finales.
Su mensaje es el siguiente:
¡¡¡ Por favor estén alertas a cualquier cosa anormal, y sean persistentes en buscar ayuda enseguida!!!
La enfermedad de Paget:
Esta es una rara forma de cáncer de mama, y aparece en la parte exterior del seno, en el pezón, la areola.
Puede parecer una erupción, que luego se convierte en una lesión con una orilla exterior reseca.
Yo nunca hubiera sospechado que era cáncer de mama, pero era. Mi pezón nunca me pareció diferente, pero la erupción me molestaba y por eso fui al medico.
Algunas veces, me picaba y me dolía, pero fuera de eso no me molestaba. Era solamente feo y molesto, y no se aliviaba con todas las cremas recetadas por mi doctor y el dermatólogo para la dermatitis que parecía ser.
Se veían preocupados, pero no me advirtieron que podía ser canceroso. Sospecho que no hay muchas mujeres que sepan que una lesión o erupción en el pezón o una areola puedan ser cáncer de mama.
El mío comenzó como una simple roncha roja en la areola. Uno de los más grandes problemas con la enfermedad de Paget es que parecen ser síntomas inofensivos y frecuentemente se confunde con una inflamación o infección de la piel, dilatando así la detección y su tratamiento.
¿Cuales son los síntomas?
Los síntomas incluyen:
1.- Una irritación persistente, goteo y resequedad del pezón, causando comezón e irritación. (Como comenté, en mi caso no me producía mucha comezón ni tenia secreción. Sí lo sentía y tenia una costra en la orilla de un lado).
2.- Una lesión en tu pezón que no sana (la mía estaba en el área de la areola con una protuberancia en el centro del pezón).
3.- Generalmente es afectado un solo pezón. ¿Cómo se lo puede diagnosticar?
Tu médico debe hacerte un examen físico y debe sugerir una mamografía de ambos senos, haciéndolo inmediatamente.
Aún cuando la irritación y las costras parezcan dermatitis (inflamación de la piel), tu médico debe sospechar cáncer si la molestias solamente en un seno y debe ordenar, inmediatamente, una biopsia de la lesión para confirmar qué está pasando.
Este mensaje debe de ser tomado en serio y enviado a la mayor cantidad de personas posibles (parientes y amigas).
Puede salvar la vida de alguien. Mi cáncer de mama se extendió y pasó a mis huesos después de recibir megadosis de quimioterapia, veintiocho (28) tratamientos de radiación y tomar Tamaxofin.
Si esto hubiera sido diagnosticado como cáncer de mama desde un principio, probablemente no se hubiera extendido.
A todos los lectores: esto es triste. Como mujeres no sabemos de enfermedad de Paget.
Si pasando esto por e-mail, podemos hacer que otras conozcan esto, y su potencial peligro, estamos ayudando a mujeres en todos lados.
Por favor: Si puedes, perder un momento y envía esto a la mayor cantidad de personas posible, especialmente a tu familia y amigas; solo toma un momento y los resultados
Pueden salvar vidas.
Pueden encontrar más información en esta página:
http://www.cirugest.com/revisiones/cir09-06/09-06-09.htm
Esta es una información a tener en cuenta por nosotras las mujeres. Creo que es importante que la divulguemos entre nuestras amigas.
En noviembre, la hermana de una amiga desarrolló una erupción en su mama, similar a la que sufren las madres jóvenes que están amamantando.
Debido a que su mamografía salió limpia, el doctor la trató con antibióticos contra infección.
Después de dos tratamientos, empezó a empeorar. Su médico le mandó hacer otra mamografía y esta vez se apreció una masa cuya biopsia reveló un tumor maligno de rápido crecimiento.
Primero le ordenaron quimioterapia para reducir el crecimiento. Luego siguió una mastectomía y después un tratamiento completo de quimioterapia, seguido de radiación.....
Nueve meses después de intenso tratamiento, fue dada de alta.
Después de un año de vivir cada día al máximo posible, el cáncer le tomó el hígado.
Siguió cuatro tratamientos y decidió, finalmente, que quería calidad de vida y no sufrir los efectos secundarios de la quimioterapia.
Tuvimos cinco extraordinarios meses y ella planeó todos y cada uno de los detalles de sus días finales.
Su mensaje es el siguiente:
¡¡¡ Por favor estén alertas a cualquier cosa anormal, y sean persistentes en buscar ayuda enseguida!!!
La enfermedad de Paget:
Esta es una rara forma de cáncer de mama, y aparece en la parte exterior del seno, en el pezón, la areola.
Puede parecer una erupción, que luego se convierte en una lesión con una orilla exterior reseca.
Yo nunca hubiera sospechado que era cáncer de mama, pero era. Mi pezón nunca me pareció diferente, pero la erupción me molestaba y por eso fui al medico.
Algunas veces, me picaba y me dolía, pero fuera de eso no me molestaba. Era solamente feo y molesto, y no se aliviaba con todas las cremas recetadas por mi doctor y el dermatólogo para la dermatitis que parecía ser.
Se veían preocupados, pero no me advirtieron que podía ser canceroso. Sospecho que no hay muchas mujeres que sepan que una lesión o erupción en el pezón o una areola puedan ser cáncer de mama.
El mío comenzó como una simple roncha roja en la areola. Uno de los más grandes problemas con la enfermedad de Paget es que parecen ser síntomas inofensivos y frecuentemente se confunde con una inflamación o infección de la piel, dilatando así la detección y su tratamiento.
¿Cuales son los síntomas?
Los síntomas incluyen:
1.- Una irritación persistente, goteo y resequedad del pezón, causando comezón e irritación. (Como comenté, en mi caso no me producía mucha comezón ni tenia secreción. Sí lo sentía y tenia una costra en la orilla de un lado).
2.- Una lesión en tu pezón que no sana (la mía estaba en el área de la areola con una protuberancia en el centro del pezón).
3.- Generalmente es afectado un solo pezón. ¿Cómo se lo puede diagnosticar?
Tu médico debe hacerte un examen físico y debe sugerir una mamografía de ambos senos, haciéndolo inmediatamente.
Aún cuando la irritación y las costras parezcan dermatitis (inflamación de la piel), tu médico debe sospechar cáncer si la molestias solamente en un seno y debe ordenar, inmediatamente, una biopsia de la lesión para confirmar qué está pasando.
Este mensaje debe de ser tomado en serio y enviado a la mayor cantidad de personas posibles (parientes y amigas).
Puede salvar la vida de alguien. Mi cáncer de mama se extendió y pasó a mis huesos después de recibir megadosis de quimioterapia, veintiocho (28) tratamientos de radiación y tomar Tamaxofin.
Si esto hubiera sido diagnosticado como cáncer de mama desde un principio, probablemente no se hubiera extendido.
A todos los lectores: esto es triste. Como mujeres no sabemos de enfermedad de Paget.
Si pasando esto por e-mail, podemos hacer que otras conozcan esto, y su potencial peligro, estamos ayudando a mujeres en todos lados.
Por favor: Si puedes, perder un momento y envía esto a la mayor cantidad de personas posible, especialmente a tu familia y amigas; solo toma un momento y los resultados
Pueden salvar vidas.
Pueden encontrar más información en esta página:
http://www.cirugest.com/revisiones/cir09-06/09-06-09.htm
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